Fra-1/AP-1 induces EMT in mammary epithelial cells by modulating Zeb1/2 and TGFβ expression
Por um escritor misterioso
Descrição
![Fra-1/AP-1 induces EMT in mammary epithelial cells by modulating Zeb1/2 and TGFβ expression](https://www.molbiolcell.org/cms/10.1091/mbc.12.1.27/asset/images/medium/mk0111409005.jpeg)
Transforming Growth Factor-β1 Mediates Epithelial to Mesenchymal Transdifferentiation through a RhoA-dependent Mechanism
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Genome‐wide cooperation of EMT transcription factor ZEB1 with YAP and AP‐1 in breast cancer
![Fra-1/AP-1 induces EMT in mammary epithelial cells by modulating Zeb1/2 and TGFβ expression](https://onlinelibrary.wiley.com/cms/asset/3d0af794-0c43-445c-91d6-d81bcbd727a7/mco2144-gra-0001-m.jpg?trick=1701221499957)
Epithelial–mesenchymal transition: The history, regulatory mechanism, and cancer therapeutic opportunities - Huang - 2022 - MedComm - Wiley Online Library
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Epithelial-mesenchymal transition - activating transcription factors - multifunctional regulators in cancer
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Regulation of epithelial-mesenchymal transition by tumor microenvironmental signals and its implication in cancer therapeutics - ScienceDirect
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EMT/MET plasticity in cancer and Go-or-Grow decisions in quiescence: the two sides of the same coin?, Molecular Cancer
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Isotoosendanin exerts inhibition on triple-negative breast cancer through abrogating TGF-β-induced epithelial–mesenchymal transition via directly targeting TGFβR1 - ScienceDirect
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Identification of EMT signaling cross-talk and gene regulatory networks by single-cell RNA sequencing
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Epithelial-mesenchymal plasticity through loss of CTCF motif accessibility and protein expression
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Inter- and intra-tumor heterogeneity in EMT states confers functional diversity in cells that lead collective invasion
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